The acute effects of antidepressant treatments on brain norepinephrine
(NE) and serotonin (5-HT) systems cannot account fully for their delayed
therapeutic action. The effects of
long-term antidepressant treatment on biogenic amine metabolism and on various
indexes of presynaptic and postsynaptic receptor function. In contrast to
variable effects on NE and 5-HT turnover and on presynaptic receptor Doxepin treatments
produce consistent alterations in a number of measures of postsynaptic amine
receptor sensitivity. Longterm treatment of Doxepin has been found to reduce β-adrenergic
sensitivity while enhancing responses to serotonergic and α-adrenergic
stimulation, suggesting that modulation of receptor sensitivity may be a
mechanism of action common to tricyclic antidepressants, "atypical"
antidepressants, monoamine oxidase inhibitors, and electroconvulsive therapy.
REFERENCE-KD
TRIPATHI, JAYPEE BROTHERS MEDICAL PUBLISHERS PVT.LTD 7TH EDITION,
REPRINT 2001,PG NO.:458,465.
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